Articles

NCOA4 maintains murine erythropoiesis via cell autonomous and non-autonomous mechanisms

Division of Genomic Stability and DNA Repair, Department of Radiation Oncology, Dana-Farber Cancer Institute, Boston, MA, USA
Division of Genomic Stability and DNA Repair, Department of Radiation Oncology, Dana-Farber Cancer Institute, Boston, MA, USA
Division of Genomic Stability and DNA Repair, Department of Radiation Oncology, Dana-Farber Cancer Institute, Boston, MA, USA
Department of Biostatistics and Translational Medicine, Medical University of Lodz, Poland;Postgraduate School of Molecular Medicine, Medical University of Warsaw, Poland
Division of Genomic Stability and DNA Repair, Department of Radiation Oncology, Dana-Farber Cancer Institute, Boston, MA, USA;Department of Cell Biology, Harvard Medical School, Boston, MA, USA
Division of Genomic Stability and DNA Repair, Department of Radiation Oncology, Dana-Farber Cancer Institute, Boston, MA, USA
Department of Pathology, Boston Children’s Hospital and Harvard Medical School, Boston, MA, USA
Department of Pathology, Boston Children’s Hospital and Harvard Medical School, Boston, MA, USA
Division of Genomic Stability and DNA Repair, Department of Radiation Oncology, Dana-Farber Cancer Institute, Boston, MA, USA;Department of Biostatistics and Translational Medicine, Medical University of Lodz, Poland
Department of Cell Biology, Harvard Medical School, Boston, MA, USA
Department of Radiation Oncology, Perlmutter Cancer Center, New York University School of Medicine, New York, NY, USA
Division of Genomic Stability and DNA Repair, Department of Radiation Oncology, Dana-Farber Cancer Institute, Boston, MA, USA
Vol. 104 No. 7 (2019): July, 2019 https://doi.org/10.3324/haematol.2018.204123